Please use this identifier to cite or link to this item: https://hdl.handle.net/10923/19010
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dc.contributor.authorOdorcyk FK-
dc.contributor.authorDuran-Carabali LE-
dc.contributor.authorRocha DS-
dc.contributor.authorSanches E-
dc.contributor.authorMartini AP-
dc.contributor.authorVenturin G-
dc.contributor.authorSamuel Greggio-
dc.contributor.authorJaderson Costa da Costa-
dc.contributor.authorKucharski LC-
dc.contributor.authorEDUARDO R ZIMMER-
dc.contributor.authorNetto CA.-
dc.date.accessioned2021-10-20T14:06:07Z-
dc.date.available2021-10-20T14:06:07Z-
dc.date.issued2020-
dc.identifier.issn0014-4886-
dc.identifier.urihttps://hdl.handle.net/10923/19010-
dc.language.isopt_BR-
dc.relation.ispartofEXPERIMENTAL NEUROLOGY-
dc.rightsopenAccess-
dc.titleDifferential glucose and beta-hydroxybutyrate metabolism confers an intrinsic neuroprotection to the immature brain in a rat model of neonatal hypoxia ischemia-
dc.typeArticle-
dc.date.updated2021-10-20T14:06:06Z-
dc.identifier.doiDOI:10.1016/j.expneurol.2020.113317-
dc.jtitleEXPERIMENTAL NEUROLOGY-
dc.volume330-
dc.spage113317-
Appears in Collections:Artigo de Periódico



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